Baltimore, July 31, 2002: The prestigious peer reviewed journal Autoimmunity published data this week by Dr. J. Bart Classen, an immunologist at Classen Immunotherapies, and David Carey Classen, an infectious disease specialist at the University of Utah, proving a causal relationship between the hemophilus vaccine and the development of insulin dependent diabetes. The data is particular disturbing because it indicates the risks of the vaccine exceeds the benefit. The findings are expected to allow may diabetics to receive compensation for their injuries and lead to safer immunization.

The study followed over 100,000 children which had been randomized in a large clinical trial to receive 1 or 4 doses of the hemophilus vaccine and over 100,000 unvaccinated children. After 7 years the group receiving 4 doses of the vaccine had an statistically significant 26% elevated rate of diabetes, or an extra 54 cases/100,000 children, compared to children who did not receive the vaccine. By contrast immunization against hemophilus is expected to prevent only 7 deaths and 7 to 26 cases of permanent disability per 100,000 children immunized. The study showed that almost all of the extra cases of diabetes caused by the vaccine occurred between 3-4 years after vaccination. Furthermore the paper provides new data proving the vaccine causes diabetes in mice and reviews data from 3 smaller human studies, which all had similar results to the current study, but were too small to reach statistical significance.

"Our results conclusively prove there is a causal relationship between immunization schedules and diabetes. We believe immunization schedules can be made safer" stated Dr. Bart Classen.

The Classens' research is already becoming widely accepted. An independent group of researchers working at a prestigious Swedish medical center recently published a paper (Ann. N.Y. Acad Sci. 958: 293-296, 2002) supporting their findings. Last year doctors attending an conference of the American College for Advancement in Medicine overwhelmingly agreed that vaccines can cause chronic diseases such as diabetes. For the latest information on the effects of vaccines on insulin dependent diabetes and other autoimmune diseases visit the Vaccine Safety Web site (http://vaccines.net) .
 



Hemophilus Meningitis Vaccine Proven to Cause Diabetes in Clinical Trial of Over 100,000 Children; Many Diabetics Eligible for Compensation

BALTIMORE, July 31 /PRNewswire/ -- The prestigious peer reviewed journal Autoimmunity published data this week by Dr. J. Bart Classen, an immunologist at Classen Immunotherapies, and David Carey Classen, an infectious disease specialist at the University of Utah, proving a causal relationship between the hemophilus vaccine and the development of insulin dependent diabetes.  The data is particularly disturbing because it indicates the risks of the vaccine exceeds the benefit.  The findings are expected to allow may diabetics to receive compensation for their injuries and lead to safer immunization.

The study followed over 100,000 children which had been randomized in a large clinical trial to receive 1 or 4 doses of the hemophilus vaccine and over 100,000 unvaccinated children.  After 7 years the group receiving 4 doses of the vaccine had a statistically significant 26% elevated rate of diabetes, or an extra 54 cases/100,000 children, compared to children who did not receive the vaccine.  By contrast immunization against hemophilus is expected to prevent only 7 deaths and 7 to 26 cases of permanent disability per 100,000 children immunized.  The study showed that almost all of the extra cases of diabetes caused by the vaccine occurred between 3-4 years after vaccination.  Furthermore the paper provides new data proving the vaccine causes diabetes in mice and reviews data from 3 smaller human studies, which all had similar results to the current study, but were too small to reach statistical significance.

"Our results conclusively prove there is a causal relationship between immunization schedules and diabetes.  We believe immunization schedules can be made safer," stated Dr. Bart Classen.

The Classens' research is already becoming widely accepted.  An independent group of researchers working at a prestigious Swedish medical center recently published a paper (Ann. N.Y. Acad Sci.  958: 293-296, 2002) supporting their findings.  Last year doctors attending a conference of the American College for Advancement in Medicine overwhelmingly agreed that vaccines can cause chronic diseases such as diabetes.  For the latest information on the effects of vaccines on insulin dependent diabetes and other autoimmune diseases visit the Vaccine Safety Web site (http://vaccines.net) . 

Classen Immunotherapies, Inc.



http://apnews.excite.com/article/20030413/D7QCOHE00.html>
\

More Children Getting Adult Diabetes
Apr 13, 11:56 AM (ET)

By DANIEL Q.  HANEY BOSTON (AP) - Once a true medical oddity, children with adult diabetes are becoming commonplace.  Doctors blame the twin evils of too much food and too little exercise and fear a tragic upswing in disastrous diabetic complications as this overweight generation reaches adulthood. At hospitals everywhere, boys and girls who range from chubby to hugely obese are being diagnosed in unprecedented numbers with type 2 diabetes.   Most are barely into their teens.  Some are as young as 6. This disease used to be called adult-onset diabetes, since it rarely occurred before middle age.  But over the past decade, it has slowly become clear this is now a disease of the young, as well.

Just how frequently is uncertain, since nationwide statistics are still being gathered.  Nevertheless, doctors are convinced they see the leading edge of a dangerous shift, one that will inevitably lead to kidney failure, blindness, heart attacks, amputations and more as these young people live another 10 or 20 years with their diabetes. "There is an epidemic of type 2 diabetes in youth, absolutely," says Dr.    Lori Laffel, head of the pediatric unit at Boston's Joslin Diabetes Center.  "Over the years, we always saw an occasional child with type 2.  It was a handful a year."

But in the early '90s, the pattern changed.  Out of the blue, it seemed, large children, usually accompanied by overweight parents, began to show up with type 2 diabetes.  During the early '90s, Joslin's totals increased five times.  Since then, they have doubled again. Until this shift, almost all diabetes in children was type 1, what was called juvenile diabetes.  In many ways, it is a different disease. Type 1 results from a misguided attack by the immune system on the insulin-making cells in the pancreas.  Victims stop making insulin completely, so they cannot convert sugar to energy. But type 2 has always been a disease of people in their 50s, 60s and beyond.  Their bodies still make insulin, just not enough.  They may go for years without realizing they have it. Around the country, doctors say Joslin's 10-fold rise in childhood type 2 over the past decade is typical.  Is it because they are looking harder or doing a better job of separating type 2 from type 1?  Not  likely, they say.

"It's not because we missed a lot of cases," says Dr.  Phillip Lee, head of pediatric endocrinology at UCLA.  "We just didn't see it.  Now referrals of type 2 are almost 50 percent of our diabetes cases." Why?  Doctors have little doubt.  They blame inactivity and overeating.   The hours on end in front of the tube, for instance, and the 20-ounce sodas in school vending machines. (Swilling four of those a day is nothing special for many kids, one doctor notes, and adds up to 1,000 calories, close to half of a boy's daily needs, all from sugar.) Virtually all children with type 2 are overweight, although this hardly  makes them unique.  The Centers for Disease Control and Prevention recently estimated that 15 percent of all U.S.  children and teenagers - and nearly a quarter of black and Hispanic youngsters - weigh too much.

Still, the ones with diabetes tend to be especially big, tall for their age and large all over.  Twelve-year-olds weigh 250 pounds.  Invariably their parents are heavy, too. "These are not little kids," says Dr.  Morey Haymond, head of diabetes care at Texas Children's Hospital.  "Even the 6-year-old is a big kid for his age." Nearly all of them have a skin condition called acanthosis nigricans, velvety, dirty-looking dark patches around the neck and other skin folds.  They are a sign of insulin resistance, an inability to respond efficiently to insulin, which is common in the overweight. In girls, the insulin resistance often triggers hormone upsets that result in facial hair, acne and ovarian cysts.

Young blacks and Hispanics have more of this kind of diabetes than do whites.  Their extra weight alone may explain some of it, although experts believe these minorities also may have an extra genetic tendency toward diabetes that is compounded by their weight. Many who do not have outright diabetes still have abnormalities that put them at high risk for diabetes.  Doctors estimate that for every youngster with type 2, four or five others have what's called syndrome X  or metabolic syndrome, a combination of obesity, insulin resistance, bad cholesterol counts, high triglycerides and high blood pressure. "Unless we make a significant alteration in their lifestyles, they will likely progress to a deteriorating course of insulin resistance, pre-diabetes and diabetes," says Dr.  Francine Kaufman, endocrinology chief at Children's Hospital Los Angeles and president of the American Diabetes Association.

Adults with type 2 diabetes face many complications that shorten or worsen their lives, although these can be reduced or prevented with medicines, weight loss and exercise. Since the problem is so new, no one knows exactly what will befall those who start the disease in their teens rather than their 50s.  But many fear the complications will emerge in early adulthood. "The horizon is really dark," says Dr.  Jorge Calles-Escandon, a Wake Forest University endocrinologist.  "We know what happens to adults with type 2 diabetes who don't take care of it properly.  They die prematurely.  They have heart attacks, strokes, blindness, renal failure.  There is no reason to believe this will be different for adolescents."

One follow-up study suggests he is right.  Researchers from the University of Manitoba tracked down 51 people, mostly in their 20s and 30s, who had been diagnosed with type 2 diabetes as children.  Two had died on kidney dialysis, and three others were still on it.  One 26-year-old woman had lost a toe to amputation, while another had gone blind. In an attempt to find out how many young Americans actually have the disease, the CDC will count all the diabetes cases among 6 million people under age 20 for five years.  It expects 6,000 of them to have it at the start and 800 more to be diagnosed annually, 30 percent of them type 2.

"Whatever we get will be an underestimate of the true disease," says the CDC's Dr.  Desmond Williams, since the study will not go looking for youngsters with diabetes, only record those who come to doctors' attention. The diabetes association recommends testing children for type 2 diabetes if they are overweight and have two other risk factors, such as a parent with the disease, signs of insulin resistance or if they are black, Hispanic or American Indian. Nevertheless, outside of big hospital obesity clinics, experts say this kind of testing is rare.  Pediatricians and family physicians simply are not trained to think about adult diabetes in the young, and without obvious symptoms, children are likely to be missed.

"It's possible that we are looking at the tip of the iceberg, the ones with the most severe disease or with pediatricians who are attuned to looking for this," says Dr.  Stephen Daniels, a pediatric cardiologist at Children's Hospital Medical Center in Cincinnati.  if so, what can be done?  Doctors understand what they are up against - a culture of cheap, high-calorie food and sedentary pleasures - and can think of no easy solutions.  These are, after all, teenagers.  They feel immortal and are loathe to do anything their friends do not, such as ordering a salad or joining an aerobics class. Some wonder if refocusing the exercise and eating habits of the entire society is the only solution.  Others think about more focused approaches, such as luring overweight girls into after-school workouts, where they might learn something both fun and sweaty, like break dancing.

Some experts believe exercise is as crucial as weight control in preventing type 2.  Pediatrics professor Bernard Gutin of the Medical College of Georgia notes that physical activity stimulates movement of sugar into cells and improves the body's response to insulin. "Vigorous exercise in kids is especially important," says Gutin, who is testing the effects of basketball, soccer and dancing on overweight boys and girls.  "That translates simply into running rather than walking." But the first, and often biggest, hurdle is getting youngsters and their parents to acknowledge they actually have a problem, that the weight must go.  Often they come from families where everyone is overweight, where nobody exercises in any way. "It's difficult to change what you have grown to accept as normal all your life, and you are surrounded with an environment that is not willing to change," says Dr.  Silva Arslanian, director of clinical research at Children's Hospital of Pittsburgh.

One large federally financed study, involving several thousand young people in North Carolina, Texas and California, will see if modest changes at school can make a difference.  It will test whether healthier choices in the cafeteria, better access to water and improved phys ed programs, among other things, have an effect on youngsters' risk of type 2 diabetes. "Can we really reverse things?" asks Kaufman, who's directing that study.  "We're not going back to the 1950s, but we could make this a healthier time in which to live."
 



More Proof Published that Common Childhood Vaccines are the Cause of the Diabetes
http://boston.com/dailynews/147/economy/_More_Proof_Published_that_Com:.shtml
P.R.Newswire, 5/27/2003 09:18
Diabetes Epidemic, Data Identifies Children Eligible for Compensation

BALTIMORE, May 27 /PRNewswire/ -- The prestigious peer reviewed journal, Journal of Pediatric Endocrinology and Metabolism published a study this week by Dr. J. Bart Classen, an immunologist at Classen Immunotherapies, and David Carey Classen, an infectious disease specialist at the University of Utah, providing support for a causal relationship between several common pediatric vaccines and the development of insulin dependent diabetes. Their previously published work proved the hemophilus vaccine, a common pediatric vaccine, caused a 25% rise in insulin dependent diabetes in children under the age of 7. Classen's research indicates most cases of diabetes caused by vaccines occur between 24 to 48 months after immunization of young children but the delay can be shorter in older children with prior damage to their pancreas. The time delay between vaccination and diabetes corresponds exactly to work from several independent groups which showed a similar delay between the initiation of autoimmunity to the insulin secreting islet cells and the development of diabetes.

"Our results conclusively prove there is a causal relationship between immunization schedules and diabetes. We believe immunization schedules can be made safer," stated Dr. Bart Classen. "Our findings help identify those who have been injured by vaccines and are eligible for compensation."Parents who think their children may have developed diabetes or any other autoimmune disease from vaccines must file a claim with the US government within 3 years of the onset of the disease in order to ensure eligibility for compensation. There is generally no cost for filing a claim.

The Classens' research has become widely accepted. For the latest information on the effects of vaccines on insulin dependent diabetes and other autoimmune diseases visit the Vaccine Safety Web site
(http://vaccines.net) .

Classen Immunotherapies, Inc.

6517 Montrose Avenue Baltimore, MD 21212 U.S.A.

Tel: (410) 377-4549 Fax: (410) 377-8526

Classen@vaccines.net http://vaccines.net SOURCE Classen Immunotherapies,
Inc.
 



By Janet McConnaughey
The Associated Press

NEW ORLEANS -- One in three U.S. children born in 2000 will become diabetic unless many more people start eating less and exercising more, a scientist with the Centers for Disease Control and Prevention warns. The odds are worse for black and Latino children: nearly half of them are likely to develop the disease, said K.M. Venkat Narayan, a diabetes epidemiologist at the CDC. "The fact that the diabetes epidemic has been raging has been well known to us for several years. But looking at the risk in these terms was very shocking to us," Narayan said.
 
The 33 percent lifetime risk is about triple the American Diabetes Association's current estimate. The implications are frightening. Diabetes leads to a host of problems, including blindness, kidney failure, amputation and heart disease, and diabetics are getting younger and younger. Including undiagnosed cases, authorities believe about 17 million Americans, nearly 6 percent of the U.S. population, have diabetes today.
 
If the CDC predictions are accurate, between 45 million and 50 million U.S. residents could have diabetes by 2050, said Kevin McKinney, director of the adult clinical endocrinological unit at the University of Texas Medical Center in Galveston. "There is no way that the medical community could keep up with that," he said. McKinney, who was not part of the study, said Narayan's procedures are valid and the estimates, presented Saturday to the American Diabetes Association, are all too likely. Diabetes, a disease associated with obesity and lack of exercise, has been an increasing worry for decades. From the mid-1960s to the mid-'90s, the number of cases tripled.The number of U.S. diagnosed cases rose by nearly half in the past 10 years, hitting 11 million in 2000, and is expected to rise an additional 165 percent by 2050, to 29 million, an earlier CDC study by Narayan and others found.

"These estimates I am giving you now are probably quite conservative," Narayan said before the diabetes association's annual scientific meeting in New Orleans. Narayan said it would be difficult to say whether undiagnosed cases would rise at the same rate. If they did, that could push the 2050 figure to 40 million or more. Doctors had known for some time that Type 2 diabetes -- what used to be called adult-onset diabetes because it typically showed up in middle-aged people -- is on the rise, and that patients are getting younger.

Nobody else had crunched the numbers to look at current odds of getting the disease, Narayan said. Overall, he said, 39 percent of the girls who now are healthy 2 1/2- to 3-year-olds and 33 percent of the boys are likely to develop diabetes, he said. For Latino children, the odds are closer to one in two: 53 percent of the girls and 45 percent of the boys. The numbers are about 49 percent and 40 percent for black girls and boys, and 31 percent and 27 percent for white girls and boys, respectively. To reach his estimates, Narayan used data from the annual National Health Interview Survey of about 360,000 people from 1984-2000, from the U.S. Census Bureau and from a previous study of diabetes as a cause of death.

Globally, the World Health Organization has estimated that by 2025, the number of people with diabetes worldwide will more than double, from 140 million to 300 million. "They estimated that by 2025, there would be close to 60 million people with diabetes in India alone. That's about the size of Great Britain or France," Narayan said. It doesn't have to happen.

Type 2 diabetes can be prevented or delayed by losing weight, exercising and following a sensible diet. A study two years ago found that walking 30 minutes a day most days of the week and losing a little weight helped the people most likely to get it cut their risk 58 percent.
 



Ann N Y Acad Sci. 2003 Nov; 1005: 404-8.  Related Articles, Links 

 
Vaccinations may induce diabetes-related autoantibodies in one-year-old children.

Wahlberg J, Fredriksson J, Vaarala O, Ludvigsson J; Abis Study Group.

Division of Pediatrics, Department of Molecular and Clinical Medicine, Faculty of Health Sciences, Linkoping University, Linkoping, Sweden.

Vaccinations have been discussed as one among many environmental candidates contributing to the immune process that later may lead to type 1 diabetes. ABIS (All Babies in Southeast Sweden) is a prospective cohort study following a nonselected birth cohort of general population. In a randomly selected sample collection from 4400 children, GADA and IA-2A have been determined at the age of 1 year. The information on vaccinations was collected from questionnaires answered by the parents and was related to beta cell autoantibodies. When studying the induction of autoantibodies using the autoantibody level of 90th percentile as cutoff level, hemophilus influenza B (HIB) vaccination appeared to be a risk factor for IA-2A [OR 5.9 (CI 1.4-24.4; p = 0.01)] and for GADA [OR 3.4 (CI 1.1-10.8; p = 0.04)] in logistic regression analyses. Furthermore, the titers of IA-2A were significantly higher (p < 0.01 in Mann-Whitney test) in those children who had got HIB vaccination. When 99th percentile was used as cutoff to identify the children at risk of type 1 diabetes, BCG vaccination was associated with increased prevalence of IA-2A (p < 0.01). We conclude that HIB vaccination may have an unspecific stimulatory polyclonal effect increasing the production of GADA and IA-2A. This might be of importance under circumstances when the beta cell-related immune response is activated by other mechanisms.

PMID: 14679101 [PubMed - indexed for MEDLINE]
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_ui
ds=14679101&dopt=Abstract
 



http://www.medicalposting.ca/children/article.jsp?
content=20040311_082809_63
68&topStory=y

    
  2004-03-11  
  Vaccine-in-a-patch linked to diabetes and multiple sclerosis   
   
  Ingredients called accelerants trigger immune system diseases in mice    
   
  Adhesive patches -- proposed as an easy way to vaccinate children --may increase the risk of developing diabetes and multiple sclerosis, Australian researchers suggest. The method is being tested in the United States, where its developers contend seven in 10 immunizations will be done this way within five years because the method offers lower cost, less pain and greater hygiene.

But research by Alan Baxter at James Cook University in Townsville, Australia, and Tony Basten of the University of Sydney raises doubts about the safety of the technique. The patches contain components of either a virus or bacteria, as do existing vaccines. But they also use accelerants that increase the body's response to the vaccine. "We've found that the accelerant also accelerates other ongoing tissue damage which may be occurring in the person," Baxter says.

Their research showed that multiple sclerosis and type 1 diabetes appeared at an earlier stage in mice treated with the accelerant than in other mice. In type 1 diabetes, the body's immune system mistakenly attacks the cells in the pancreas that produce insulin. In multiple sclerosis, it attacks the myelin sheath covering nerves in the brain and spinal cord.  We found that the way this accelerant increases a person's immune response to the vaccine appears to be exactly the same as the mechanism by which it increases the reaction to your own tissue, and this effect cannot be separated."

Baxter says the findings mean patch vaccines will need to be tested much more extensively than planned.
 



http://archpedi.ama-assn.org/cgi/content/abstract/158/11/1031

Safety of the Trivalent Inactivated Influenza Vaccine Among Children A
Population-Based Study

Eric K. France, MD, MSPH; Jason M. Glanz, MS; Stanley Xu, PhD; Robert L.
Davis, MD, MPH; Steven B. Black, MD; Henry R. Shinefield, MD; Kenneth M. Zangwill,
MD; S. Michael Marcy, MD; John P. Mullooly, PhD; Lisa A. Jackson, MD, MPH;
Robert Chen, MD, MA


Arch Pediatr Adolesc Med. 2004;158:1031-1036.

Background  To our knowledge, there are no published population-based studies on the safety of the inactivated trivalent influenza vaccine among children. Objective  To screen a large population of children for evidence of increased medical visits in the 2 weeks after influenza vaccination compared with 2 control periods. Secondary analyses included shorter risk periods and restricted age categories.

Design  Self-control screening analysis. Children vaccinated from January 1, 1993, through December 31, 1999, were randomly divided into 2 equal groups. In group 1, risks of outpatient, emergency department, and inpatient visits during the 14 days after vaccination were compared with the risks of visits in 2 control periods. Significant plausible medically attended events identified in group 1 were then analyzed in group 2, using the same 2 control periods. Medically attended events significant in both groups were considered potentially associated with vaccination and were assessed by medical record review.

Setting  Five managed care organizations in the United States.

Participants  Children younger than 18 years who received an influenza vaccination in one of the managed care settings (N = 251 600). Main Outcome Measure  Among vaccinated children seen for a medically attended event, the odds of the visit occurring in the 2 weeks after vaccination vs during 1 of the 2 control periods.

Results  Study participants incurred 1165, 230, and 489 different diagnoses during the 14 days after vaccination according to the outpatient, emergency department, and inpatient data, respectively. Four diagnoses were positively associated with the vaccine in both groups 1 and 2: impetigo, dermatitis, uncomplicated diabetes mellitus, and ureteral disorder not otherwise specified. After medical record review, impetigo (9 cases) in children 6 to 23 months old remained significantly associated with vaccination.

Conclusion  This large screening safety study did not reveal any evidence of important medically attended events associated with pediatric influenza vaccination.
 

(I wonder if the parents of these children thought diabetes mellitus, ureteral disorders and impetigo was insignificant)

Author Affiliations: Clinical Research Unit, Kaiser Permanente Colorado, Denver (Drs France and Xu and Mr Glanz); Scientific Advisor to the Centers for Disease Control Vaccine Safety and Development Activities Branch (Dr Davis) and the Immunization Safety Branch (Dr Chen), Centers for Disease Control and Prevention, Atlanta, Ga; Department of Epidemiology, University of Washington, Seattle (Dr Davis); Kaiser Permanente Vaccine Study Center, Northern California Kaiser Permanente, Oakland (Drs Davis, Black, and Shinefield); UCLA Center for Vaccine Research, Torrance, Calif (Dr Zangwill); Southern California Kaiser Permanente, Panorama City (Dr Marcy); Center for Health Research, Northwest Kaiser Permanente, Portland, Ore (Dr Mullooly); and the Center for Health Studies, Group Health Cooperative, (Dr Jackson).
 



Vaccine Induced Inflammation Linked To Epidemic Of Type 2 Diabetes And Metabolic Syndrome

05 Apr 2008

Newly published data by Dr. J. Barthelow Classen in The Open Endocrinology Journal shows a 50% reduction of type 2 diabetes occurred in Japanese children following the discontinuation of a single vaccine, a vaccine to prevent tuberculosis. This decline occurred at a time when there is a global epidemic of type 2 diabetes and metabolic syndrome, which includes obesity, altered blood cholesterol levels, high blood pressure, and increased blood glucose resulting from insulin resistance.

Classen proposes a new explanation for the epidemic of both insulin dependent diabetes (type 1 diabetes), which has previously been shown to be caused by vaccines and non insulin dependent diabetes (type 2 diabetes). Upon receipt of vaccines or other strong immune stimulants some individuals develop a hyperactive immune system leading to autoimmune destruction of insulin secreting cells. Other individuals produce increased cortisol, an immune suppressing hormone, to suppress the vaccine induced inflammation. The increased cortisol leads to type 2 diabetes and metabolic syndrome. Japanese children have increased cortisol secretion following immunization compared to White children and this explains why Japanese have a relative high rate of type 2 diabetes but low rate of insulin dependent diabetes compared to Whites. The lower cortisol response attributed to type 1 diabetes and the higher cortisol response attributed to type 2 diabetes explains why type 1 diabetics are generally leaner than type 2 diabetics since elevated cortisol causes weight gain.

"The current data shows that vaccines are much more dangerous than the public is lead to believe and adequate testing has never been performed even in healthy subjects to indicate that there is an overall improvement in health from immunization. The current practice of vaccinating diabetics as well as their close family members is a very risky practice," says Dr. J. Barthelow Classen.

Classen's research has become widely accepted. To view the published papers and to find out the latest information on the effects of vaccines on autoimmune diseases including insulin dependent diabetes visit the Vaccine Safety Web site http://www.vaccines.net/newpage11.htm

Classen Immunotherapies, Inc.
http://www.vaccines.net